Pigeon Herpesvirus (PHV)
Attacks the upper respiratory tract and conjunctiva, causing breathing difficulty and ocular discharge; spreads quickly in densely housed lofts.
Spread by respiratory droplets, ocular and nasal secretions and contaminated equipment; latently infected pigeons can reactivate the virus and shed it after stress.
Causes upper respiratory inflammation and conjunctivitis, impairing respiratory function and vision and reducing racing condition; secondary infection can worsen the disease and prolong the course.
Distinguishes herpesvirus infection from other respiratory pathogens, guides antiviral and symptomatic treatment, assesses the carriage rate in the flock, and reduces flock outbreaks under dense housing.
Must be differentiated from respiratory pathogens such as Mycoplasma and Newcastle disease; herpesvirus mainly causes conjunctivitis and upper respiratory signs, and its hallmark is reactivation of latent infection after stress.
Incubation period 2–7 days; latently infected pigeons can reactivate and shed virus after stress such as transport or racing.
Collect throat and conjunctival swabs for qPCR detection of viral nucleic acid for confirmation, interpreted together with the upper respiratory and conjunctival signs.
Treatment is mainly symptomatic and supportive: control secondary bacterial infection, reduce stress and isolate affected birds. Specific medication must be decided by a veterinarian according to the case.
Keep the loft well ventilated, control stocking density, isolate affected birds and disinfect regularly.
The health knowledge base carries the full entry for Pigeon Herpesvirus (PHV) — pathogen characteristics, transmission and impact, clinical signs, and diagnostic and control guidance.
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